Quantitative investigations of speech production deficits are reported in three aphasic patients. Two had impaired paraphasic performance in repetition tasks but relatively well preserved spontaneous speech (conduction aphasia). The other patient had impaired paraphasic spontaneous speech but intact repetition (transcortical motor aphasia). In repetition tasks which required active semantic processing the conduction aphasics were facilitated and the transcortical motor aphasic impaired; in tasks which required passive repetition the opposite pattern of dissociation was observed. These findings are accounted for within a two-route model of the speech production process.

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