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Impact Factor
4.159
SI: Endocrinology and Metabolism
30 out of 133

Editors-in-Chief
Andrea C. Gore, PhD, and Stephen R. Hammes, MD, PhD

Editorial Board

About the journal

Endocrinology: Molecular and Physiological Basis of Endocrine Health and Disease is the premier basic science journal of the Endocrine Society, publishing basic and translational research at molecular, biochemical, cellular, genomic, comparative, and organismal levels since 1917.

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Featured in This Issue

Hogan et al.Markers of Islet Endothelial Dysfunction Occur in Male B6.BKS(D)-Leprdb/J Mice and May Contribute to Reduced Insulin Release

Drs. Gore and Hammes have selected additional research articles as Editor’s Choice in the latest issue of Endocrinology. Find out more.

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Endocrinology publishes advances in basic and translational research for the global endocrinology community, at molecular, biochemical, cellular, genomic, comparative, and organismal levels. Submit your paper today.
Free color online

Free Color Online

Present your data brilliantly. Endocrinology, the premier basic science journal, now offers free color online.   

Merger

Why did we merge?

Endocrinology and Molecular Endocrinology have merged. By combining the aim and scope of both journals, Endocrinology, celebrating 100 years, can now offer the single-most comprehensive basic science journal in hormone research.

Molecular Endocrinology archives

Editor's Choice

en.2016-1468

Circulating Insulin-Like Growth Factor I Regulates Its Receptor in the Brain of Male Mice

The activity-dependent entrance of serum IGF-I into the brain results in tight modulation of brain IGF-I receptor levels to spatio-temporally restrict its own actions in the brain. Read more.

News & Views: Spatiotemporal Regulation of Insulin-Like Growth Factor-1 and Its Receptor in the Brain: Is There a Role for Growth Hormone?

en.2016-1645

A Thalamo-Hypothalamic Pathway That Activates Oxytocin Neurons in Social Contexts in Female Rats

A group of posterior thalamic neurons is activated by pups in mothers and also in familiar females. These neurons are excitatory, receive ascending afferent connections, and project to the PVN to innervate oxytocin neurons. Read more.

News & Views: Who Are You and Where Am I? New Insights Into How Animals Determine Their Social Context

en.2016-1340

The Steroidogenic Acute Regulatory Protein (StAR) Is Regulated by the H19/let-7 Axis

Overexpression of the lncRNA H19 stimulates StAR expression by antagonizing let-7 (which itself inhibits StAR at the posttranscriptional level). These results uncover a novel mechanism underlying the regulation of StAR expression and represent the first example of lncRNA-mediated control of the rate-limiting step of steroidogenesis. Read more.

News & Views: Yet Another Scenario in the Regulation of the Steroidogenic Acute Regulatory (STAR) Protein Gene

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COPE

Committee on Publication Ethics

Endocrinology is a member of and subscribes to the principles of the Committee on Publication Ethics (COPE)
Resource Identification Initiative

Advancing Research Resource Identifiers (RRIDs)

Endocrinology supports the aims and recommended practices of the Resource Identification Initiative.

CME

CME

Endocrinology reviewers are eligible for Continuing Medical Education credits.

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